Cystometric, Sphincter and Electromyelographic Abnormalities in Parkinson’s Disease

Jens T. Andersen, William E. Bradley

Research output: Contribution to journalArticle

40 Citations (Scopus)

Abstract

Bladder function was studied in 24 patients with a diagnosis of parkinsonism using cystometry, sphincter electromyography, flowmetry and electromyelography. A high incidence of disturbances in detrusor function and sphincter control was documented and the signal tracing studies showed prolonged conduction times, giving evidence of peripheral neuropathy. The abnormalities of detrusor function were ascribed to the lesion of the basal ganglia, whereas the sphincter disturbances indicate impairment of the corticospinal tract as a result of Parkinson’s disease.
Original languageEnglish
Pages (from-to)75-78
Number of pages4
JournalJournal of Urology
Volume116
Issue number1
DOIs
Publication statusPublished - 1976

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Pyramidal Tracts
Rheology
Parkinsonian Disorders
Electromyography
Peripheral Nervous System Diseases
Basal Ganglia
Parkinson Disease
Urinary Bladder
Incidence

Cite this

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title = "Cystometric, Sphincter and Electromyelographic Abnormalities in Parkinson’s Disease",
abstract = "Bladder function was studied in 24 patients with a diagnosis of parkinsonism using cystometry, sphincter electromyography, flowmetry and electromyelography. A high incidence of disturbances in detrusor function and sphincter control was documented and the signal tracing studies showed prolonged conduction times, giving evidence of peripheral neuropathy. The abnormalities of detrusor function were ascribed to the lesion of the basal ganglia, whereas the sphincter disturbances indicate impairment of the corticospinal tract as a result of Parkinson’s disease.",
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Cystometric, Sphincter and Electromyelographic Abnormalities in Parkinson’s Disease. / Andersen, Jens T.; Bradley, William E.

In: Journal of Urology, Vol. 116, No. 1, 1976, p. 75-78.

Research output: Contribution to journalArticle

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AB - Bladder function was studied in 24 patients with a diagnosis of parkinsonism using cystometry, sphincter electromyography, flowmetry and electromyelography. A high incidence of disturbances in detrusor function and sphincter control was documented and the signal tracing studies showed prolonged conduction times, giving evidence of peripheral neuropathy. The abnormalities of detrusor function were ascribed to the lesion of the basal ganglia, whereas the sphincter disturbances indicate impairment of the corticospinal tract as a result of Parkinson’s disease.

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